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Kidney Patients Harmed

December 18th, 2011 No comments

Water fluoridation at ordinary EPA allowed levels worsens arthritis, including crippling arthritis. See:

Critique of EPA’s Maximum Contaminant Level Goal (MCLG) for Fluoride

Michael Connett, Ellen Connett, Paul Connett, Chris Neurath
Fluoride Action Network (FAN)
December 16, 2005

 

Research Should Be Done In Now Non-Fluoridated Calgary

October 16th, 2011 No comments

10-16-11

With the cessation of fluoridation with silicofluoride, lead levels should have dropped in water and in blood.

And kidney patients in the dialysis centers should be doing better; kidney function should be improving.

How would you go about getting someone in Calgary to look into these issues? Who might I ask?

If we fail to do this research, we will be missing a golden opportunity. This is a biological experiment made available by a mass change in an environmental condition. It would be much easier to study than looking for a few people who moved into or out of fluoridated and non-fluoridated areas.

See: http://fluoride-class-action.com/hhs/comments-re-lead

We quote[56] from Fluoride and Lead by Frances Frech:

Let us tell you a tale of two cities–Tacoma, Washington, and Thurmont, Maryland. Both of them saw significant decline in [blood] lead levels only six months after fluoridation was stopped. (In Tacoma, that was due to equipment problems, in Thurmont, it was a temporary ban by the city council.) Tacoma registered a drop of nearly 50% …; in Thurmont it was 78%. To the best of our knowledge, no other explanations were offered. In Thurmont the ban is now permanent.”

Unfortunately, Tacoma returned to fluoridating its drinking water and a battle continues over whether to reverse this policy.

 ***

From: James Beck, University of Calgary
Sent: Sunday, October 16, 2011 2:50 PM
To: James Robert Deal
Subject: Re: Compare lead levels in water and lead levels in Calgary – before and after cessation of fluoridation

Mr. Deal:

I agree with you that the suggested investigation would be important if competently done. As I said I will see if I can find an appropriate, and approachable, investigator. You ask how you would find such a person and whom you might ask. If you want to try this directly yourself (I see no objection to that, but I have never heard of such a move in the past) then I suggest you focus on a nephrologist. Preferably an active experimental researcher. If there isn’t such a creature in Calgary, then look for a clinical nephrologist. One can ask, say, the head of the Department of Medicine for a name and contact information or ask the Dean of Medicine. Here’s a start: http://medicine.ucalgary.ca/ . I’ve just made a futile and time-consuming effort to get the specific possible contacts for you and I have a commitment to meet now.

I suppose you realize that getting such a project going would take considerable time. That’s not a reason not to try of course.

 

Jim Beck

***

Dr. Beck,

Many cities have terminated fluoridation. Calgary could be the template for investigating the changes that result in personal health.

If you could find someone there, it would be your good deed for the year

James

 

Categories: Canada, Kidneys, Lead Tags: ,

Saurheber: Poisoning from Fluoridated Public Water

March 1st, 2010 1 comment

Chemical Analysis of Poisoning from Fluoridated Public Water
Richard D. Sauerheber, Ph.D.

Abstract. The mechanism by which fluoride from unnatural sources exerts adverse effects in man and animals is examined. Low level artificial fluoridation of municipal water can cause well known alterations in teeth and bone structure with incorporation of fluoride into a wide array of tissues and increased hip fracture tendency, depending on years of exposure and water hardness. High accidental levels cause acute lethal poisoning and are responsible for fluoride listings on poisons registries and for their major industrial use as rodenticides, insecticides or pediculicides. Solubility calculations indicate that blood fluoride concentrations required to decrease calcium below normal physiological levels compare to those present in the tissues of poisoned victims and to those causing decreased beat rates in isolated heart cells in culture. The effects of calcium ion and pH over broad ranges on the free fluoride ion concentration were determined. Acute lethal poisoning with heart attack, and also many of the chronic ‘low’ level effects of fluoride, are mediated by calcium binding by fluoride ion. At a pH typical of gastric juice, approximately 50% of fluoride is protonated as hydrofluoric acid HF, with 50% remaining the free fluoride ion. The significance of these observations is discussed in terms of potential hazards, both short and long term, associated with consumption of artificially fluoridated waters of varying calcium content.

Read the full article here:
http://fluoride-class-action.com/wp-content/uploads/sauerheber-chemical-analysis-of-poisoning-from-fluoridated-public-water.pdf

Fluoride & Our Kidneys

February 22nd, 2010 No comments

Fluoride and Kidneys

Fluoride plays perhaps more mischief with the kidneys than with any other organ.

See this excellent summary of the literature by Carol Clinch.

“One of the strongest physiological effects of fluorides in drinking water (e.g. hydrofluorosilicic acid) is in the kidney, a point to consider in light of increased rates of kidney failure during recent decades.

http://fluoride-class-action.com/wp-content/uploads/carol-clinch-2009-fluoride-and-kidneys.pdf

Kidney disease markedly increases an individual’s susceptibility to fluoride toxicity. In healthy adults, the kidneys are able to excrete approximately 50% of an ingested dose of fluoride. However, in adults with kidney disease the kidneys may excrete as little as 10 to 20% and young children may only excrete 15% of an ingested dose – thus increasing the body burden of fluoride and increasing an individual’s susceptibility to fluoride poisoning (e.g. renal osteodystrophy). 

Categories: Fluoride Articles, Kidneys Tags: